So don't go wiring a night scope and a targeting laser and recognition grip onto your Sparq gun and then come complaining to me for a refund when you short out the power cell. Let me repeat that Modification voids your warranty! For example, powered implants and energy weapons can be recharged to a higher capacity with a higher maintenance skill. The maintenance skill also has subtle benefits when using devices. Maintenance tools exist that allow you to improve the condition of a weapon.
You can determine the current condition of a piece of equipment by moving the cursor over the equipment's icon in your inventory when the weapon is equipped, the ammo box displays a coloured dot and number corresponding to the condition. The results implicate vasopressin in the maintenance of cardiovascular function during the early phase of shock induced by endotoxin, and demonstrate the ability of corticosteroid to substantially improve cardiovascular integrity in the absence of vasopressin.As your maintenance skill increases, you can keep your weapons in better shape. Respiration rates for the untreated B were severely depressed until death after endotoxin, whereas the SD and treated B remained at or above control. Heart rates in the SD increased sharply after endotoxin and continued to rise, whereas both treated and untreated B increased slowly. All treated animals survived this period. Corticosteroid treatment (300 mg/kg) administered 30 min before endotoxin in the B prevented the severe decrease in blood pressure with values falling to and remaining near 70 mm Hg for the 240-min observation period. In contrast, the blood pressure response of the SD dropped to 70 mm Hg but recovered to values near control until sacrificed 240 min after endotoxin. This resulted in death for all ten animals within 3 1/2 hours eight of ten had expired within 90 min. Every weapon in System Shock 2, with the exception of the melee weapons and the Psi Amp, has a maintenance level. Endotoxin in the B caused an immediate drop in blood pressure to below 40 mm Hg with no recovery. After recovery from anesthesia, endotoxin (20 mg/kg) was administered. Conscious, unrestrained male Sprague-Dawley rats (SD) and male homozygous Brattleboro rats (B) lacking vasopressin were anesthetized with 2% enflurane and cannulas were placed in the carotid artery and jugular vein. The influence of vasopressin on the maintenance of cardiovascular and respiratory function during early endotoxin shock was evaluated.
This study was conducted as an initial step to elucidate the role of vasopressin in the events leading to irreversible shock.
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